Alzheimer s Disease Detected By Alzheimer Mouse Models

Alzheimer’s disease Detected in an Earlier Stage The biological field is in constant investigation and there are new findings all the time. Science is so exciting that writers, educators and publishers want to make it available and understandable to everyone. Being this the case, people generate easy accessible websites with the intention to inspire others to acquire knowledge of what’s new in science, so they include the new findings, most of the time from journal reports of PhD students or a team of researchers have done. An example of these types of websites is â€Å"iflscience.com,† where I found an article called â€Å"Destruction of Brain Cell Connections May Be the Earliest Indicator of Alzheimer’s† about a new discover that could help in the†¦show more content†¦The topics include: Alzheimer’s disease and what provokes it, amyloid-beta proteins, synapses, synaptic pruning, neurons, cognitive and microglial cells. Alzheimer’s disease affects mostly individuals in their mid-6 0s and is â€Å"a type of dementia that causes problems with memory, thinking and behavior† (Alzheimer s Association). The articles reflect the discovery of the Alzheimer’s disease affects the brain by the destruction of the connection of the brain cells which causes damage in neurons and accelerate cognitive decline, and occasionally call it the earliest stage. Amyloid-beta is a term that refers to the plaques made by the proteins which interferes and cause the loss of synapses also known as synaptic pruning is the loss of connections between neurons caused microglia, that according with The Campbell Biology In Focus textbook, â€Å"microglia are immune cells that protect against pathogens† (Campbell.) The authors of the research support â€Å"microglia that prune excess synapses in development are inappropriately activated and mediate synapse loss in Alzheimer’s disease.† (Taub, 2016) The prior terms explain what happen with the brain in that early stage, that after this research, it is able to identify. The researchers describe in the journal article the use of C1q, â€Å"the initiating protein of the classical complement cascade, is increased and associated with synapses before overt plaque deposition† (Hong et al, 2016). Incorporation of